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These results may explain the ineffectiveness of the 10 M dose of LA treatment, as the increased formation of peptide fragments (particularly LA fragment 2) may share enough similarities to the Zot and zonulin receptor-binding motif (G XXX VQ X G) to competitively inhibit LA binding to EGFR and/or PAR2

This isn't unique to GHK-Cu

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The main functions and features of the most prominent pro-angiogenic factors are discussed briefly below

doi: 10.1021/tx960145c
