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Hyperglycemia causes metabolic programming and epigenetic modifications, which sustain the overexpression of the pro-oxidant enzymes (PKC, p47phox, and mitochondrial adaptor p66Shc) and reduce the levels of antioxidant enzymes like SOD even after normalizing glucose levels

Chen W, Su H, Feng L, Zheng X (2016) Andrographolide suppresses preadipocytes proliferation through glutathione antioxidant systems abrogation

Mitochondrial transplantation improves anxiety-and depression-like behaviors in aged stress-exposed rats

Discriminatory Detection of Cysteine and Homocysteine Based on Dialdehyde-Functionalized Aggregation-Induced Emission Fluorophores

American heart journal 2009