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Zhang H, Limphong P, Pieper J, Liu Q, Rodesch CK, Christians E, and Benjamin IJ

[246] screened the Q-Marker of this drug based on the property-response-component ternary relationship

Glutathione oxidation as a trigger of mitochondrial depolarization and oscillation in intact hearts

Exogenous GSH (0.5 mM) ameliorated onion bulb epidermis damage, improved membrane permeability, prevented protoplasmic swelling, and maintained cell viability under salt stress (150 mM NaCl)

Use adaptive dosing and staging matched to disease phase and compartment: for example, early ischemiareperfusion may benefit from acute, time-boxed control of mitochondrial bursts and labile iron to limit Fenton chemistry, while chronic vascular or organ remodeling calls for rebalancing NOX activity and ER stress together with lifestyle re-entrainment (exercise, nutrition, sleep, exposure control) to reset redox tone without erasing physiological H 2 O 2 signalling (Poznyak et al