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Description
The Kat6a null allele was generated as described previously, with the deletion of 5 exons (exons 48 of 17 exons in NM_001364449.1 and exons 59 of 18 exons in NM_001081149.2) resulting in a premature stop codon in the catalytic domain (Voss et al, 2009)
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ESPEN 60, 122134
Secondly, this study was modeled on daily L-C supplements for three months only
showed that the blockage of CPT1A (with etomoxir) and the lipid synthesis/lipolysis (with orlistat) decreased the viability of the androgen-dependent prostate cell lines LNCaP, VCaP, and patient-derived prostate cancer cells 34
