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The therapeutic effect included the succinylcholine-induced behavioral agitation, muscle twitches, and motionless resting, and completely eliminated post-succinylcholine hyperalgesia, immediately eliminated leg contractures (intramuscular succinylcholine), counteracted both edema and the decrease in muscle fibers in the diaphragm and injected/non-injected anterior tibial muscles) [68]

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Furthermore, the study hypothesizes a potential interaction of AOD9604 with the beta-adrenergic pathway, especially concerning the beta(3)-adrenergic receptors (beta(3)-AR), which are considered to be key lipolytic receptors found in fat cells