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Injection of A42 into rat basal forebrain followed a week later by examination of oxidative damage in hippocampus demonstrated a significant elevated level of HNE-modified proteins (13), consistent with the known diffusion of HNE from its site of origin in cell or organelle membranes to other areas within the cell (8, 34) and consistent with the connection between the forebrain and outer molecular layer of hippocampus (98)

(7, 8) During the oxidation of acetaldehyde into acetate by the aldehyde dehydrogenase enzyme (ALDH), excess NADH is produced which alters the proportion of NADH to NAD

Inflammatory bowel disease: an overview of immune mechanisms and biological treatments

Mol Membr Biol 17:143156

A hypothetical astrocytemicroglia lactate shuttle derived from a 1H NMR metabolomics analysis of cerebrospinal fluid from a cohort of South African children with tuberculous meningitis