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1) is consistent with both the conversion pathway of mercapturic acid 34 and the remarkable decrease of hepatic GSH in 1,2-DCP-administered rats 11 described in previous reports

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As confirmation, we used the crisis that occurred with the high intra-abdominal pressure-induced syndrome, in which intra-abdominal hypertension simultaneously affected all abdominal vessels and organs for a considerable period and restrained the ability to recruit alternative pathways, such that a deadly situation was created before therapy initiation

Yadlapati S, Snchez-Luna SA, Gromski MA, Mulki R