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Description
Carnitine synthesis defect and autism With the overall goal to identify exonic copy number variants in the genome of ASD patients, Celestino-Soper and coauthors[42] analyzed 3743 samples for detecting disease-causing copy number variants (CNVs) that are not detected by most techniques used in conventional research and clinical diagnosis laboratories
Additionally, epigenetic regulation through DNA methylation and histone modifications may underlie increased long-term susceptibility to changes in bone and BMAT (193)
Varicocoele-associated male infertility: cellular and molecular perspectives of pathophysiology
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