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[DOI] [PubMed] [Google Scholar] Brammer L

They demonstrated that pretreatment of cultured SOD1 G93A astrocytes with MitoQ was able to reduce the formation of superoxide and peroxynitrite radicals, protect motor neurons against oxidative damage and improve the ability of mitochondria to generate ATP (Cassina et al., G93A mouse model, treatment with MitoQ at the onset of disease also delayed the decline of mitochondrial function in the spinal cord, protected against motor neuron loss and astrogliosis in the spinal cord

Reprod BioMed Online (2004) 8:37684

Cellular Recovery: Promotes repair of damaged DNA, contributing to overall skin health

Thus, a better molecular understanding of signals and mediators in regenerative tissue responses and resolution of metabolic injuries is critical to develop clinically-relevant therapeutic interventions focusing on the enforcement of epithelial regenerative capacity by improving metabolic fitness, a novel strategy for combating intestinal diseases