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Mammals, in particular, have developed biochemical and metabolic pathways to control pathogen infection by increasing amino acid catabolism to aid immune responses, thus restricting the availability of nitrogen-containing nutrients to invading microorganisms [1]

It is described as protecting the liver against environmental toxins by detoxifying amine byproducts of protein metabolism, and its richest dietary source is lecithin (phosphatidylcholine), found in egg yolks and soybeans

Specifically, the binding of IGFBP3 to GRP78 increases autophagic site formation and autophagic system flux, thereby promoting breast cancer cell survival even under glucose starvation and hypoxic conditions (57)