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Telomere dysfunction induces metabolic and mitochondrial compromise
Intestinal lesions produced by prednisolone: prevention (cytoprotection) by 16,16-dimethyl prostaglandin E2
Research suggests that it may be associated with cytoprotective mechanisms and tissue repair processes in preclinical musculoskeletal, gastrointestinal, neurologic, and vascular injury models
Pathogen induced changes in antioxidant enzyme levels have been shown in a number of plants (Table 3)
