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While LDL is mostly taken up by cells in target organs, excess LDL can deposit in the endothelial wall of blood vessels and initiate a cascade that ultimately contributes to the formation of atherosclerotic plaques [16, 17]
Impaired germinal center reaction in mice with short telomeres
(2020) investigated the interaction of GSH with hydrogen sulfide in alleviating salinity stress
[57] In neuronal progenitor cells, DNA damage is associated with increased secretion of amyloid beta proteins A40 and A42
