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Impaired mitochondrial oxidative phosphorylation limits the self-renewal of T cells exposed to persistent antigen

The effective blood-brain barrier mechanism impeding the transport of monoamines has been attributed to both the presence of tight junctions cementing adjacent endothelial cells of the cerebral capillary walls, along with high concentrations of dopa decarboxylase and monoamine oxidase (MAO) in the cerebral capillary endothelium (Bertler et al, 1966

Under conditions of fasting, hepatic metabolism is shunted toward gluconeogenesis, making fewer glucose precursors available for glucuronidation

359 The diminished efficacy of HSCs can be attributed not only to telomere shortening but also to an increased risk of DNA damage 360 and elevated levels of ROS production

PMID 19289127