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In ovarian cancer cells, vitamin K2 inhibited ovarian cancer cell growth by disturbing the TR3/Nur77 signaling pathway, which is involved in cell survival and death regulation

Modifying the TME is a complex process

G6PD-deficient variants that do not result in CNSHA are designated class II or class III, depending on the severity of the reduction in enzyme activity in red blood cells

Therefore, a vicious cycle is created in the ovarian epithelium, resulting in the formation of cysts, low-quality ovarian follicles, and infertility [13]

The spectra were centroided and de-isotoped by Analyst software (ABSciex, Toronto, Canada)
