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Interestingly, age-related elevations in the plasma CXCL9 and KTR were dramatically suppressed in the ERGO group (Figs

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Many have reported a reversibility of LCAC-mediated effects in ischaemia following normoxic reperfusion, suggesting direct enzymatic or ionic modulation by LCACs ( First described by max , and RMP dose-dependently (75300 M) and with an effect latency analogous to that found in the ischaemic myocardium (210 min)