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Mitochondrial dysfunction serves as a critical amplifier: excessive glucose oxidation leads to a burst production of mitochondrial ROS, which directly damages glomerular endothelial cells and podocytes while oxidatively modifying Toll-like receptor 4 (TLR4), enhancing macrophage sensitivity to pathogen-associated molecular patterns (PAMPs) and triggering sterile inflammatory responses (32)

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The role of macrophages in right ventricular remodeling in experimental pulmonary hypertension

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84 :1509-18 Corresponding authors: Yunfei Ling, Associate Professor of Cardiovascular Surgery, Department of Cardiovascular Surgery, West China Hospital of Sichuan University, No