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The most well-established mechanisms include through (1) NF-B inhibition, (2) suppression of STING signaling, (3) interference of pro-inflammatory cytokine transcription, and (4) through NRF2 target gene signaling pathways

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Ramanathan R, Ali AH, Ibdah JA (2022) Mitochondrial dysfunction plays central role in nonalcoholic fatty liver disease
Role of the mucins in pathogenesis of COPD: implications for therapy

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