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Research consistently shows that the biological window for neuroplasticity can be extended by peptide interventions, but translating biological potential into functional recovery still requires active engagement with the tasks and skills being recovered

Studies suggest that various molecular pathways are involved in IC/BPS, including the JUN N-terminal kinase (JNK) pathway, transient receptor potential (TRP) channels, activated mast cells, mucosal signalling, circadian rhythm regulation, inflammation and neural dysregulation

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When readers encounter bold claims about longevity peptides, asking Which rung of the ladder does this sit on? is often more useful than asking whether the molecule sounds impressive