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Antipurinergic interventions based on suramin administration appear to act by inhibiting the excessive signaling of ATP and related nucleotides released from stressed cells

The dual action creates a net negative fat balance: more fat is broken down, less new fat is formed (Ng et al., 2000)

The peptide modulates nitric oxide synthesis, which governs vascular tone, blood flow, and inflammatory responses across gut, muscle, and neural tissue

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Finding out how CnoX recognizes its substrates and what is the role, if any, played by the TPR domain in controlling substrate selectivity are other outstanding questions