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Given that dietary FA are re-esterified and packed as VLDL-TG, it is possible that dietary FA spillover contributes to the postprandial rise in circulating liver-derived VLDL

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Also the observation that HeLa cells have a significant fraction of PS with limited mobility, and that cortical actin contributes to the confinement of PS in the plasma membrane 68 suggests that immobilisation of a fraction of PS could be due to binding of other cytosolic proteins as well