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Description
A high Notch pathway activation predicts response to -secretase inhibitors in proneural subtype of glioma tumor-initiating cells
Eprenetapopt is converted to the Michael acceptor methylene quinuclidinone (MQ), which inhibits the antioxidant metabolite GSH (glutathione) by reacting with its thiol groups 14
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influenzae both colonize the nasopharynx, and they are able to cause invasive disease including meningitis and septicemia
All GSTs have a basic protein fold comprising two subunits with C-terminal and N-terminal domains
