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177 Other conditions with similar clinical features include thyroid disease, hyperprolactinemia (high levels of the hormone prolactin), an androgen-producing tumor of the ovary or adrenal gland, and non-classical congenital adrenal hyperplasia (NC-CAH), which causes adrenal steroid 21-hydroxylase deficiency

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doi: 10.1007/s40620-020-00789-y 21 FitzGeraldG

The kinetic data can be best explained by a ternary complex in which PfGrx recruits and activates GSH at the 1st glutathione-interaction site to facilitate the rate-limiting glutathionylation of roGFP2 WT (S 2 ) and its subsequent deglutathionylation in accordance with monothiol mechanism (iii) from Fig