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Description
Therefore, potential molecular mechanisms leading to the deregulation of FXR and PPAR activation possibly arising as a consequence of altered UGT1A activity in htgUGT1A -SNP mice are discussed and compared to the results of human population studies observed during NAFLD and NASH
Animal models are valuable for identifying mechanisms and generating hypotheses, but they do not always translate directly to human outcomes
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Cell Biol Toxicol 35 (5), 407421
Some of the studies carried out to date are reported in Table 7
