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Moreover, ferroptotic stress may not only contribute to tissue damage but also release proinflammatory mediators and damage-associated molecular patterns (DAMPs), reinforcing inflammation and accelerating tissue degeneration

In a cross-over design study we observed recoveries of excreted sulforaphane metabolites (principally sulforaphane-N-acetylcysteine) to be about 5% when a glucoraphanin-rich beverage was administered with a range of 1 to 45% but about 70% with a much narrow range of inter-individual variation when a sulforaphane-rich beverage was used [68]

[Google Scholar] [PubMed] [CrossRef] Ghaneifar Z, Yousefi Z, Tajik F, Nikfar B, Ghalibafan F, Abdollahi E, Momtazi-Borojeni AA (2020)

Nat Rev Clin Oncol (2016) 13:11932

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