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Microglial activation and tau burden predict cognitive decline in Alzheimer s disease
[DOI] [PMC free article] [PubMed] [Google Scholar] 24.Meldolesi J
doi: 10.3389/fnins.2019.00721
The intricate balance between providing a substrate for normal cellular functions and potentially supporting malignant cells underscores the importance of careful consideration and individualized approaches in the clinical application of glutamine supplementation
Keap1 is a cysteine-rich protein that can be oxidized by ROS and results in conformational change, which in turn liberates NRF2
