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Golgi stress mediates redox imbalance and ferroptosis in human cells

Additionally, one study indicated that HMGB1 can upregulate heme oxygenase 2 and transferrin expression through the RAS-JNK/p38 pathway, promoting erastin-induced ferroptosis [101]

Histone lactylation: from tumor lactate metabolism to epigenetic regulation

Although residues C32 and C88 of PfGrx are redox-inactive during the monothiol mechanism, they both facilitate the deglutathionylation of GSSR substrates

Boeckman Jr., and Mary H
