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He has over 30 years of experience in medtech, healthcare, technology, and software as a service industry sector
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Concurrently, oscillatory shear stress activates the TRPM2 channel, mediating cytosolic Ca 2+ influx and NFAT signaling, which drives VSMC transition from a contractile to synthetic phenotype, promoting migration and proliferation and accelerating plaque formation [120,121]
Bibcode:2001PlMed..67..306D
