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This observation together with largely cancer cell-specific expression of pmCiC provides a mechanistic explanation for why gluconate could be partially responsible for the increased anti-cancer effects observed when used with other therapeutics, like Zn 2+ and Ca +2

Ratner RE, Dickey R, Fineman M, et al

The levels of the lipid peroxidation product, MDA, in the liver, reflect the oxidative stress and GSH consumptions

This process is non-specific and can lead to the hyper-glutathionylation of proteins, altering their activity

U373 and HMC3 cells were treated with ferrostatin-1 (Fer-1) (10 M) or a vehicle (dimethyl sulfoxide [DMSO]), followed by infection with HSV-1 (MOI = 0.1)