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Under physiological conditions, KEAP1 mediates NRF2s constitutive proteasomal degradation (McMahon et al., 2003) while BACH1 acts as transcriptional antagonist of NRF2s target genes (Zhang and Hannink, 2003)

Laboratory studies investigate how the analogue may modulate neuronal growth, survival, and synaptic plasticity under various stress or oxidative conditions

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We further showed that FUS/TLS, U1-snRNP and CPSF-160 may indeed form a large complex (~1.8 MDa)

Similarly, compared to iRPMI alone, addition of Albumax to iRPMI decreased PS exposure and hemolysis levels, though not significantly
