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Under physiological conditions, KEAP1 mediates NRF2s constitutive proteasomal degradation (McMahon et al., 2003) while BACH1 acts as transcriptional antagonist of NRF2s target genes (Zhang and Hannink, 2003)

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Laboratory studies investigate how the analogue may modulate neuronal growth, survival, and synaptic plasticity under various stress or oxidative conditions

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We further showed that FUS/TLS, U1-snRNP and CPSF-160 may indeed form a large complex (~1.8 MDa)

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Similarly, compared to iRPMI alone, addition of Albumax to iRPMI decreased PS exposure and hemolysis levels, though not significantly

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