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Kidney diseases, specifically acute kidney injury (AKI) and chronic kidney disease (CKD), represent major global health burdens.3,4 The pathophysiology of these renal diseases involves interconnected mechanisms, notably inflammation, fibrosis, and oxidative stress, with the excessive generation of reactive oxygen species playing a critical role in disease initiation and progression.5,6 Consequently, therapeutic strategies that modulate oxidative stress and associated redox-sensitive signaling pathways are increasingly relevant to nephrological research and clinical practice

189,190 Finally, the diol 10( E ),12( Z ),14( E )-9( S ),16( S )-DiHOTrE has been identified after further oxidation of 9( S )-HpOTrE (an ALA-metabolite of plant 5-LOX) by human 15-LOX-2

We also found that exposure of hepatocytes isolated from mice drinking H-HRW to palmitate overload demonstrated a protective effect from H 2 by reducing hepatocyte lipid accumulation in comparison to mice drinking regular water

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Methyl jasmonate alleviates arsenic-induced oxidative damage and modulates the ascorbateglutathione cycle in oilseed rape roots