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In tissues, the ketosteroid isomerase reactions catalyzed by GSTs are strictly dependent on dehydrogenases such as 3-hydroxysteroid dehydrogenase and 17-hydroxysteroid dehydrogenase for production of the 3-keto substrates from the corresponding 3-hydroxysteroid precursors, since the GSTs are lacking dehydrogenase activity

Moreover, tumor-targeted stimuli-responsive nanocarriers that release cargo in response to tumor-specific stimuli like pH, redox potential, or enzyme can be used to deliver the systemic side effects [189, 364]

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Oral Dis 2010

In vivo studies demonstrated that 75 exhibited significant hyperuricemic and serum XO inhibitory effects in mouse models of potassium oxonateinduced hyperuricemia