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Concurrently, ER can induce autophagy independently of classical pathways (mTOR, PI3KCIII, MAPK) via regulators such as BAG3, ATG7, and WIPI, while ER modulates autophagy in a context-dependent manner, influencing autolysosome formation, cyclin degradation, HIF function, and interacting with autophagy-related proteins (ULK1, BECN1, LC3B, ATG16L1, p62) and the NLRP6 inflammasome

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Metabolites of GRP produced by microbiota can be categorized into the following three groups: (i) GLs such as glucoerucin, (ii) ITCs such as SFN and erucin, and (iii) ITC-nitriles such as SFN-nitrile and erucin-nitrile