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Clinical trials showed that exogenous BDNF had limited success as therapeutic, mainly due to suboptimal pharmacokinetic properties and side effects
cellulose-based membranes provoke complement activation and monocyte IL-6 release, thereby exacerbating DKD-related inflammaging (130)
It is becoming increasingly clear that there is a massive energetic deficiency in AD brains that is paired with mitochondrial dysfunction, it is possible that such pathologies and loss of function precede the actual neuronal death itself
A 5mg vial with 2mL water creates 2.5mg/mL concentration
doi: 10.1016/j.brainres.2008.05.067
