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Further studies revealed that Sirt5 plays an important role in supporting cancer cell metabolism by regulating various enzyme activities and protecting glutaminase C (GAC), an enzyme essential for glutamine catabolism, from degradation

PloS ONE 9 , e83979 (2014)

These contribute to the activation of various regulated cell death mechanisms, including ferroptosis, which has been implicated in the pathogenesis of IBD (5,6)

Dephosphorylation of the protein at methionine and histidine residues is required for its external release from ATP7A (9, 11)

[DOI] [PMC free article] [PubMed] [Google Scholar] [22].Forman HJ, Zhang H, Rinna A