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Description
Multi-omics data reveal that SAA1 + fibroblasts exacerbate periodontitis by regulating macrophage inflammation and chemotaxis
[7] CROT activity has also been reported in mouse liver, kidney, adipocyte, mammary gland, skeletal muscle, and heart tissues
Consequently, intracellular Fe 2+ could not be oxidized to Fe 3+ and excreted from macrophages via FPN, which may be the potential mechanism of iron retention in plaques
KGF, keratinocyte growth factor
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