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Since the GPX4-catalyzed reduction of lipid peroxides is disabled by glutathione deprivation, elevated ChaC1 may play a role in ferroptosis by degrading glutathione [109]

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For this reason, it is believed that expression in a eukaryotic system of a complete ORF encoding an intact HEV gene would result in production of a protein which could form HEV-virus-like particles

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The main consequence of RBC rupture is the release of intracellular content, in particular Hb and arginase 1