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Ferroptosis regulation within mitochondria Compelling evidence supports the importance of mitochondria in driving CDI ferroptosis, hence it is reasonable to assume that there might be mitochondria-localized defense systems to prevent ferroptosis

o , Number of mass spectrometry runs, left, and number of MS/MS spectra per run, right, in the human blood metabolome meta-analysis dataset

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In addition, fibroblasts from individuals affected by HGPS, or from mice lacking ZmPSTe24, demonstrate a marked delay in the recruitment of p53 binding protein 1 (53BP1) to sites of DNA repair upon exposure to DSB-inducing irradiation (Liu et al., 2005)