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Description
As seen in Fig 1A although the levels of miR-21-5p, serving as a control, were unaffected by starvation, miR-4488 levels increased by 4 h of GS starvation but declined progressively at 24 and 48 h (Fig 1B)
doi: 10.2337/dc23-S006 153
doi: 10.1128/JB.05105-11
A central mechanism driving such pathological scarring is the persistent activation of the transforming growth factor-beta (TGF- )/Smad signaling pathway, which continuously stimulates fibroblasts and myofibroblasts, resulting in excessive collagen deposition and fibrotic remodeling (31, 32)
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