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How is Vitamin B12 used
The possibility that sustained folic acid intake would deplete vitamin B-12 independent of hematopoiesis suggests a plausible alternative mechanism based on the known details of the renal handling of holoTC, whereby folic acid might interfere with holoTC reuptake or cause its sequestration in the kidney, promoting the excretion of holoTC and further depleting metabolically active vitamin B-12 during vitamin B-12 deficiency
International journal of clinical pharmacology research
Inhibition by 5Amino1MQ has been used in research to probe links between methylation status, redox state, and lipid metabolism
It is observed that infants, children, adolescents, and women of reproductive age are also at high risk of deficiency
