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HSP32/HO-1 is the primary mediator of celastrol's protective effect
Depuydt et al., 2009)
Visceral fat is strongly associated with increased cardiovascular disease risk, and its reduction typically correlates with improved cardiovascular health markers
doi: 10.1007/s12011-012-9563-x
249 This activation promotes catabolic processes that generate energy and, concurrently, inhibits mTORC1, halting energy-consuming processes such as gluconeogenesis, lipid synthesis, and protein synthesis
