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All mice were euthanized on day 15
Elucidating the detailed molecular mechanisms by which AMPARs traffic to and away from synapses is an active topic of current investigations (see West and Greenberg 2012)
[96] Adlercreutz H, Honjo H, Higashi A, et al
Deficiency in the level of activity of glucose-6-phosphate dehydrogenase (G6PD) is the basis of favism, primaquine (an anti-malarial drug) sensitivity and several other drug-sensitive hemolytic anemias, anemia and jaundice in the newborn, and chronic nonspherocytic hemolytic anemia
7 BladtF.Friese-HamimM.IhlingC.WilmC.BlaukatA
