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The next day, the sections were treated with biotinylated secondary antibody (1:1,000) and incubated in avidinbiotinperoxidase complex for 1 h

[180]demonstrated that in MASLD, compromised GSH-GPX4 axis and decreased lipophilic antioxidants enhance ferroptosis, resulting in hepatic mitochondrial dysfunction

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2 (download XLSX Statistical source data

Taken together, these results suggest regulation of GS may also be important in determining the overall GSH synthetic capacity under certain conditions and especially in non-hepatic tissues