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In mouse models of sepsis, itaconate and its derivative, 4-octyl itaconate (4-OI), inhibits STING phosphorylation and the production of downstream inflammatory factors like IFN- and TNF- through alkylation of STING at cysteine sites 65, 71, 88, and 147 to ameliorate inflammation [152]

Additionally, research professionals have demonstrated that DSIP administration can influence the release of pituitary hormones, such as adrenocorticotropic hormone (ACTH) and luteinizing hormone, both of which play a critical role in the stress response and regulating metabolism

Integr Med (Encinitas) 2017;16:4249

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