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Update on Antiepileptic Drugs 2019
Preclinically, these inhibitors reduce infarct volumes and improve functional recovery, not merely by lowering lactate but by reprogramming cellular metabolism: forcing neurons to resurrect oxidative phosphorylation in salvageable penumbral regions and redirecting glucose toward the PPP to bolster NADPH-dependent antioxidant defenses [208, 209]
Autophagy and its link to cardiac metabolism in HF Metabolic remodeling in HF is also associated with changes in autophagy
Aung Phyu
Can J Physiol Pharmacol
