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Interleukin-6 and ARDS Progression to ARDS in a subset of COVID-19 patients can be partially attributed to dysregulation of host cytokine production precipitating a positive feedback cycle of inflammatory insult and parenchymal injury

As in the thapsigargin untreated condition, the insulin secretagogue effect was reduced in the PDIA6 A5 mutant compared with the WT (Fig

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