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Overall, the benefits of aerobic exercise are limited by the functional reserve beyond the mutation load in MM patients, and the magnitude of improvement is largely determined by the underlying genotype ( MM patients experience exercise intolerance and delayed post-exercise recovery as a result of defective oxidative phosphorylation, which limits ATP production ( MM patients typically exhibit elevated oxidative stress due to ETC defects, resulting in excessive ROS production that damages mtDNA

HMB levels decline with age, and this drop is linked to diminished lean muscle mass and strength, which can be complemented through supplementation

Fish Diseases, 35, 799-808

Bassareo V, Frau R, Maccioni R, Caboni P, Manis C, Peana AT, et al

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