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The proposed mechanism involves controlling glutamate release in the synaptic cleft, modulating NMDA receptor stimulation, affecting excitatory amino acid transporter (EAAT)-mediated reuptake, and maintaining the glutamate/glutamine cycle within astrocytes

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Mice deficient in ABHD15 are unresponsive to the effects of insulin on lipolysis (122, 123)

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The legality depends on the specific peptide and its intended use

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A meta-analysis found that higher dietary intake of these vitamins was inversely associated with depression risk, with significant effects observed in women

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