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Description
However, the sheer volume of ROS generated during a prolonged illness rapidly depletes the body's intracellular glutathione stores
This idea was further supported when similar phenotypes were observed across ferroptosis-sensitive and ferroptosis-resistant cancer cell lines (Extended Data Fig
In the glutathione system, it is what drives the conversion of GSSG back to GSH
In this context, increased expression of detoxifying enzymes may lead to the development of redox adaptative mechanisms that could perturb cell homeostasis and even promote leukemic transformation
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