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Controlling membrane cholesterol content

Abnormalities in apoC3 and apoA5 were associated with different forms of HTG, such as familial hyperchylomicronemia [38, 40], familial combined hyperlipidemia [41,42,43], and familial dysbetalipoproteniemia [44]

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Neuroinflammation, oxidative Stress, and neurogenesis in a mouse model of chronic fatigue Syndrome, and the treatment with Kampo medicine

(2013, Nature Medicine) found that L-carnitine is metabolized by intestinal microbiota to trimethylamine-N-oxide (TMAO), a proatherogenic substance associated with increased CVD risk