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Modulation of dopamine D1 [239], D2 [240], and GABA [241], receptors by Hg, may also involve Hg-thiol group interaction, although the particular target Cys residues have yet to be identified (Table 1)

We further demonstrate that activation of both mTOR-STAT3 and ERK-CREB pathways, induced by elevated circulation amino acids, is responsible for upregulation of Vgf and Gcg expression following glucagon receptor blockade

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Chunbao Li, Nanjing Agricultural University, China Updates Copyright 2020 Zhang, Qi, Zeng, Bao, Chen, Liao, Wei, Cao, Huang, Luo, Jiang and Mo